Tyrosine kinase inhibitorPhase 3 trialInvestigational

Sotorasib

How it works

Blocks the KRAS G12C mutation on cancer cells, preventing them from growing and dividing.

Cancer types

Lung CancerKRAS-mutated

Efficacy

In clinical trials, around 57% of KRAS G12C-mutated patients achieved an objective response, with a median progression-free survival of approximately 6.8 months.

Side effects

Moderate

Side effects can be significant and may require dose adjustments or supportive medication, but the treatment is usually continued.

Evidence from research

StudyCancer typeStageEfficacy
New Treatment for Lung Cancer: Sotorasib Shows PromiseLung CancerreviewSR has demonstrated significant objective response rates and progression-free survival benefits in pretreated patients with NSCLC.Source →
KRAS Inhibitor Resistance Linked to EGFR ActivationLung Cancerlab-studyDual blockade of KRASG12C and EGFR showed a substantial antitumor effect in a xenograft model of sotorasib-resistant NSCLC.Source →
New approaches to treating lung cancer with KRAS mutationsLung CancerreviewSource →
Combining Sotorasib with Other Treatments May Help Lung Cancer PatientsLung Cancerphase-2The patient achieved a significant and durable clinical benefit lasting 23 months.Source →
Resistance to Cancer Treatment Sotorasib in Lung Cancer PatientsLung CancerobservationalOne patient with acquired MET amplification achieved a renewed partial response to the combination of sotorasib and tepotinib after progression on sotorasib monotherapy.Source →
New Cancer Treatment Shows Promise in Advanced Colorectal CancerColorectal Cancerphase-3The overall response rates were 30.2% for sotorasib 960 mg-panitumumab, 7.5% for sotorasib 240 mg-panitumumab, and 1.9% for investigator's choice.Source →

This information is provided for general education only and is not medical advice. Always consult a qualified healthcare professional before making treatment decisions.